Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Then again, our capability to rescue SERT functionality in KI midbrain synaptoneurosomes by the inhibition of FAK implies elevated FAK signaling downstream in the Pro32Pro33 mutant, as verified by improved pFAK localization in five-HT synapses. https://kameronfcsgt.blogdigy.com/pro33-an-overview-48229879